Autism can be caused by a known genetic state, experts say.
They found that children with muscular nutrition type 1 (DM1) are 14 times more likely to cause autism spectrum disorders.
DM1 is a disabled person inherited by parents, causing gradual muscle weakness, fatigue and numerous cognitive problems.
Experts can also affect the way the brain develops and processes the initial life information, and potentially potentially changes the nerve paths related to communication, behavior and social interaction, which are characteristic of autism.
This discovery can unlock some of the mystery about how autism develops, providing an important clue to biological roots.
DM1 is caused by a defective genes, and now researchers think that genes can play a role in autism.
Researchers said the discovery is closer to understanding autism with specific and tracking origins as well as spectrum.
And decisively, it raises hope for more targeted support for patients with a state and treatment with a focus on repairing damaged genes.
But the team also stressed that DM1 is much rarely than autism, and everyone with DM1 is not diagnosed with autism.
Researchers at the University of Las Vegas (UNLV), Nevada, have found that people with muscular nutrition type 1 (DM1) may be 14 times more likely to develop autism (stock images).
In DM1, the DNA strands of DMPK repeated DNA strands, tandem repeated expansion (TRES), the genes do not function properly.
For people with DM1, this leads to symptoms such as gradual muscle weakness and involuntary exercise.
Functional errors cause protein imbalances that affect other genes responsible for brain function.
Researchers said that due to these genetic disorders, people with DM1 can develop signs of autism such as repetitive movements, lack of coordination and sensory problems.
Autism affects about 7 million Americans, but only 140,000 people are diagnosed with DM1.
Researchers at the University of Las Vegas (UNLV), Nevada, said the findings that they could easily diagnose autism of people with the same conditions as DM1 and to bring about treatment to restore damaged genes.
Dr. Ryan Yuen, a research author and senior scientist of Genetics and Genome Biology Program in Las Vegas, said: ‘Our research shows a new way to characterize the genetic development of autism.
‘By identifying the molecular path behind this connection, we can investigate the development of a new approach to the ASD diagnosis and the precision therapy that re -emits these proteins into the genome.’
This means that the essential protein far from the DNA can be added again, which can be repaired and prevents additional errors.
meantime, Another new study From China’s researchers, we found that non -invasive brain irritation treatment can improve signs of certain autism, such as sleep and social interactions.
The processing of two pulse current stimulation (TPC) includes sending an electric impulse through the electrode placed on the scalp of the patient.
The signal from the electrode is thought to increase brain activity in certain areas.
The team found that children between 3 and 14 years of age, who had received 20 TPC sessions for four weeks, received ‘important’ improvements in sleep, language, sensory problems and socialization.
According to the latest CDC data, autism is increasing autism in the United States and affects one out of 31 children. This is a huge increase in one out of 150 in the early 2000s.
Many experts believe that the rise is due to judging and diagnosis, but health authorities, such as Robert F Kennedy JR, are responsible for environmental factors such as pesticides, food additives and ultrasound scans.
Published in UNLV research Natural neuroscienceThe researchers analyzed RNA in 38 genetic sets from people with autism or without autism. RNA is a major molecule that helps cells to build proteins and functions properly.
The team found that when the DMPK gene in charge of the DM1 is repeated, it produces a ‘toxic RNA’ that binds to proteins involved in DNA production during brain development.
‘Toxic RNA’ depletes protein and does not bind other RNA molecules, causing protein imbalances and errors in other surrounding genes.
Dr. Yuen said: ‘Tre is like a sponge that absorbs all of these important proteins in the genome. Without this protein, other areas of the genome do not function properly. ‘
Researchers can cause autism that DM1, muscle-occupancy, autism can be caused by repeated DMPK genes.
Dr. Lukasz SZNAJDER, the research director and assistant professor of Las Vegas University (UNLV), said:
‘This is the way we started connecting dots. We found molecular links or overlaps, which is the key to causing autism symptoms for children with nutrition. ‘
DM1 is a life -oriented life organ, such as the heart and lungs as it leads to weakening of muscle around the limbs. This leads to abnormal heart rhythms and breathing problems.
Symptoms generally occur in adolescence or adolescence and begin with the weaknesses around the muscles of the face, neck, fingers and ankles.
This condition affects 140,000 Americans and the expectation is about 48-55 years.
The research author said that more research should connect DM1 to autism, but the group plans to see if these DNA errors occur in other genes related to autism.
