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It is difficult to catch all the potential privileges that take GLP-1 receptor agent drugs such as Ozempic. So far, researchers have found that it can help to reduce the risk of this drug. Cardiovascular disease and ColonTamped the symptoms Danang ovarian Syndrome (PCOS)Even Treat addiction. Of course, it is above the design of blood sugar control and weight loss.
More and more studies can now help GLP-1 lower the risk of Alzheimer’s disease and dementia. And there is one more study to add to the mix.
New research has been published Zama neurology There are more cases where one of these drugs has been found by discovering the connection between those who take the GLP-1 receptor agent and those who have a low risk of Alzheimer’s disease.
I think about what experts think about new research and how it applies to you.
Meet an expert: Me Ali, MDMemorialcare Surgical Correction Center of Orange Coast Medical Center in California Pound Tin Valley; Verna Porter, MDALZheimer ‘S Disease and Neurocognitive Decenders, Dementia’s Neuroscience Decenders, Dementia in Pacific Neuroscience
What did the study find?
For this study, researchers analyzed 396,963 health records in Florida from January 2014 to June 2023. All participants were over 50 years old and type 2 diabetes. At the beginning of the study, no one was diagnosed with Alzheimer’s disease and related dementia.
Researchers have found that they have a significant risk of significantly significant risk than those who lower blood sugar compared to Alzheimer’s disease and other drugs than those who lower blood sugar than Alzheimer’s disease and other drugs.
How can GLP-1 reduce the risk of Alzheimer’s?
Type 2 diabetes patients are more likely to cause Alzheimer’s disease. (research Patients with type 2 diabetes suggest that the risk of dementia is 50 % higher.)
The reason is that the risk of blood vessel damage, which is the risk factor of insulin resistance, inflammation and Alzheimer’s disease, and the GLP-1 receptor agent can lower the risk of these complications. Verna Porter, MDALZheimer ‘S Disease and Dementia’s Neuroscience Institute in Santa Monica, California, nerves and specialists and supervision of DEMENTIA.
But this drug can help people lose weight, feel better and lead a healthier lifestyle. Me Ali, MDMemorialcare Surgical Central Medical Officer of Orange Coast Medical Center in California Fountain Valley. “If the patient reduces weight, the risk factor (for Alzheimer’s disease) is as follows. Heart disease Diabetes is also improved. ”
Dr. Potter pointed out that she has a “careful optimism” of the latest research and treats many patients with type 2 diabetes and Alzheimer’s disease.
How does GLP-1 work in the brain?
GLP-1 receptor agents act in the body in several different ways. However, these drugs are less hungry, and they are less likely to overeat by sending signals to the brain.
As a result, many people lose weight to this drug. But the exact mechanism of how drugs work in doing things like food noise, addiction, and the current reduction of Alzheimer’s risks is still being investigated.
Are there any other studies to support this result?
This is not the first study to find the connection between the GLP-1 receptor agent and the alzheimer’s disease in the type 2 diabetes patients. no way study It was published in the journal in October Alzheimer’s and Dementia Those who took the Semaglutide (GLP-1 receptor agent drug) found that the risk of developing Alzheimer’s disease is lower than those who took other drugs.
Another research has been published Natural medicine In January, those who took GLP-1 receptor agent drugs found that there was a low risk of developing a lot of health, including dementia.
It is important that many studies of these studies investigate the effects of GLP-1 receptor agent on patients with type 2 diabetes. In other words, taking this medicine without type 2 diabetes is not clear whether it will affect the risk of Alzheimer’s.
However, this growing research is clearly raised as many questions and the possibility of treatment are developed.
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